The optic tract is a part of the visual system in the brain.

Information from the left visual field travels in the right optic tract.

For example, a person who has a lesion of the right optic tract will no longer see objects on his left side.

The atrophy of grey matter, the neuron bodies, is likely due to its association with the optic tract.

There, partial decussation occurs, and about 53% of the fibers cross to form the optic tracts.

A lesion in the left optic tract will cause right-sided homonomous hemianopsia.

Retinal sensors send signals through the optic tract to the Lateral geniculate nucleus.

The M-cells receive their information from the axons of parasol cells exiting the optic tract.

The two optic tracts, representing the right and left visual fields, emerge posteriorly from the posterior chiasm.

Knowing the neurocircuitry of visual signal flow through the optic tract is very important in understanding bitemporal hemianopsia.

Posterior lesions may also involve the optic tract and cause a contralateral homonymous hemianopia.

These axons form the optic nerve, optic chiasm, and optic tract.

The optic tract projects to a number of anatomically distinct regions in the brainstem, each of which makes a unique contribution to visual function.

At the gross anatomical level the optic tract can be seen to terminate in a number of discrete relays in the brainstem.

Studies found that the volume of white matter (myelinated nerve connections) was reduced in the optic tract, but not in the primary visual cortex itself.

These manifest themselves in three principal ways: chronic meningitis, involvement of the both the optic tract and eye and sensineural deafness.

Complete decussation of the optic tract has been seen as a method of ensuring the open eye strictly activates the contralateral hemisphere.

In humans the optic tract, commissure, and optic nerve bridge the substantial distance between eyes and diencephalon.

Beyond the optic chiasm, with crossed and uncrossed fibers, optic nerves become optic tracts.

Each optic tract terminates in the lateral geniculate nucleus (LGN) in the thalamus.

Several pretectal nuclei, particularly the OPN, receive illuminance information from the ipsilateral retina via the optic tract.

Vascular and neoplastic (malignant or benign tumours) lesions from the optic tract, to visual cortex can cause a contralateral homonymous hemianopsia.

He demonstrated that homonymous hemianopsia was caused by lesions in the occipital lobe and optic radiation as well as the optic tract.

Retinal sensors convey stimuli through the optic tracts to the lateral geniculate bodies, where optic radiations continue to the visual cortex.

Because the eyes no longer receive visual information, the disuse of the connected optic tract causes a loss of grey matter volume in the primary visual cortex.